Ukutya kwe-keto kuhlala kuyimpikiswano, ngaphandle kwalo lonke uphando apho. Abaninzi bayisebenzisela ukunciphisa umzimba, ngelixa abanye bekhetha ukunciphisa ukutya kwe-carbohydrate. Ukuza kuthi ga ngoku, phantse akukho mntu wayezibuza ukuba imiphumo yayiya kwahluka kumadoda namabhinqa. Kukho umahluko?
Izazinzulu zase-UC Riverside ziye zaqhuba Isifundo ukufunda indlela ukutya okuthandwayo kwe-ketogenic kunye nokukhawuleza kokuzila ukutya okusebenzayo kwinqanaba le-molecular, nokuba ngaba zombini izini ziyazuza kuzo ngokulinganayo. Ingcamango emva kokutya kwe-keto kukuba amanqanaba aphantsi e-carbohydrates kunye namazinga aphezulu kakhulu amafutha kunye neprotheni aya kunyanzela umzimba ukuba usebenzise amafutha ukuba abe namafutha, okukhokelela ekulahlekeni kobunzima.
Kwelinye icala i ukutya okukhawulezileyo isebenza kumgaqo ofanayo, ukukhawulela ukutya kwifestile encinci yexesha emini. Ngexesha leeyure ngaphandle kokutya, umzimba uchitha iivenkile zeswekile kwaye uqala ukutshisa amanqatha. Amafutha aguqulwa abe yimizimba yeketone enokuthi ingqondo isebenzise isibaso.
Ngaba yahlukile kwabasetyhini kunye namadoda?
Ngaphandle kokuthandwa kwazo, izazinzulu azikachongi imizila yemfuza okanye iiproteni ezivumela ukutya ukuba kusebenze. Ke olu phando lutsha lucinga ukuba sele uyayazi indlela abasebenza ngayo. Isitshixo sinokuthi sibe yiprotheni, ebizwa ngokuba yi-HNF4, efumaneka kumazinga aphezulu esibindi. Yinto ebhaliweyo, eguqula i-DNA ibe yi-RNA, ethi emva koko iguqulelwe kwiiprotheni ezintsha, kwaye iza ngeendlela ezimbini, i-P1 okanye i-P2.
Iingcali zenzululwazi ekuqaleni zaphanda i-P2 njengeprotheyini ye-pro-cancer. Abazange bafumane ikhonkco kumhlaza, kodwa baqaphela ukuba iimpuku ezinamazinga aphezulu e-P2 kwisibindi sabo nazo zinemizila eyahlukeneyo yemetabolism. Baye bafumanisa ukuba i-P2 ibonakala kwiimali ezinkulu kamva ngemini, enokuthi ichaze ukuba kutheni iigundane zingakhange zifumane ubunzima obuninzi ukuba ixesha lokutya lithintelwe; nokuba batye kakhulu.
Kurhanelwa ukuba i-enzayim ebuthathaka amandla inokubangela utshintsho phakathi kwe-P1 kunye ne-P2, enokuvumela inkqubo yokutshisa amafutha ukwenzela amandla. Kolu phononongo, ingqalelo ekhethekileyo ihlawulwe kwiindlela apho iigundane zamadoda nabasetyhini ziphendula kwi-ketogenic kunye nokutya okukhawulezileyo.
Ukutya amanqatha amaninzi kwenza abafazi batyebe
Ukutya kwe-keto akubonakali kusebenza kakuhle kubasetyhini, kuba Metabolise amafutha ngokwahlukileyo kwaye sinemizila eyahlukeneyo evuliwe kwaye icinyiwe ekuphenduleni ukuzila. Kodwa ngokwenene asazi ukuba kutheni okanye kwenzeka njani; yile nto sinethemba lokuyifunda.
Ukuba ukutya kuyasebenza nakweyiphi na isondo, abaphandi bayalumkisa ukuba Akukho kutya kufuneka kuthathwe ngokugqithiseleyo. Akucaci ukuba onke amanqatha afakwe kwi-metabolized kwi-keto yokutya okanye ukuzila ukutya, okanye ukuba isixa esikhulu siqokelela nje emzimbeni. Ukutya okuqhelekileyo kwaseJapan kuqulethe i-20% yamanqatha, i-American diet avareji ye-35%, kunye nokutya kwe-ketogenic kunokuquka i-70 okanye i-80%, mhlawumbi isixa esiphezulu kakhulu.
ukuba siyatya inamafutha, uya kusityebisa ekugqibeleni. Kanye njengokutya kakhulu nantoni na kuya kusenza sityebe, kuquka nezucchini. Eyona nto ibalulekileyo sisixa esikutyayo, into esiyityayo kunye nexesha losuku.